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A Person addict for alcohol gets his liv...

A Person addict for alcohol gets his liver destroyed because:

A

Liver stores excess of protein

B

liver stores excess of fat

C

Liver stores excess of starch

D

Liver stores excess of glycogen

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### Step-by-Step Solution: 1. **Understanding Alcohol Addiction and Its Effects on the Liver**: - Alcohol addiction leads to chronic consumption of alcohol, which has several detrimental effects on the liver. The liver is responsible for metabolizing alcohol, and excessive intake can overwhelm its capacity to process it. 2. **Cirrhosis and Liver Damage**: - One of the primary conditions associated with chronic alcohol consumption is cirrhosis. This is a condition characterized by the scarring (fibrosis) of liver tissue, which impairs liver function. The liver becomes inflamed due to the toxic effects of alcohol. 3. **Chronic Inflammation**: - The inflammation in the liver can be caused by several factors, including hepatitis (liver infection), exposure to toxins, and excessive alcohol consumption. In the case of alcohol, it leads to a condition known as alcoholic hepatitis. 4. **Replacement of Hepatocytes**: - Hepatocytes are the main functional cells of the liver. In cirrhosis, these cells are damaged and replaced by adipose (fat) tissue. This process is known as steatosis, where fat accumulates in the liver, leading to further liver dysfunction. 5. **Consequences of Fat Accumulation**: - As fat accumulates in the liver, it leads to a decrease in the liver's ability to perform its functions, including detoxification, protein synthesis, and production of biochemicals necessary for digestion. This accumulation ultimately results in liver failure. 6. **Conclusion**: - Therefore, a person addicted to alcohol gets his liver destroyed primarily because the liver stores excess fat due to the chronic effects of alcohol consumption, leading to cirrhosis. ### Final Answer: A person addicted to alcohol gets his liver destroyed because his liver stores excess fat, leading to cirrhosis. ---
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