Which of the following statement is incorrect?

Options

A. ADH prevents conversion of angiotensinogen in blood to angiotensin
B. Aldosterone facilitates water reabsorption
C. ANF enhances sodium reabsorption
D. Renin causes vasodilation

Explanation

To solve the question "Which of the following statement is incorrect?", we need to analyze each of the provided statements regarding hormones and their functions in the excretory system.

### Step-by-Step Solution:

1. **Identify the Statements**:
- We have four statements to evaluate. Let's denote them as:
- Statement A: ADH prevents conversion of angiotensinogen to angiotensin.
- Statement B: Aldosterone facilitates water reabsorption.
- Statement C: ANF enhances sodium reabsorption.
- Statement D: Renin causes vasodilation.

2. **Evaluate Statement A**:
- The statement claims that ADH prevents the conversion of angiotensinogen to angiotensin. This is incorrect. The conversion of angiotensinogen to angiotensin is mediated by renin, and ADH does not play a role in this conversion. Therefore, Statement A is incorrect.

3. **Evaluate Statement B**:
- Aldosterone is known to facilitate the reabsorption of sodium ions, which in turn increases osmotic pressure, leading to water reabsorption. This statement is correct.

4. **Evaluate Statement C**:
- The statement claims that ANF enhances sodium reabsorption. This is incorrect. Atrial Natriuretic Factor (ANF) actually promotes sodium excretion, not reabsorption.

5. **Evaluate Statement D**:
- The statement claims that renin causes vasodilation. This is incorrect as well. Renin acts as a vasoconstrictor, leading to the narrowing of blood vessels.

6. **Conclusion**:
- From the analysis, we find that Statements A, C, and D are incorrect. However, the question specifically asks for the incorrect statement among the options. The most clearly incorrect statement is Statement A, as it misrepresents the role of ADH.

### Final Answer:
The incorrect statement is **Statement A**: ADH prevents conversion of angiotensinogen to angiotensin.

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